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#adenocarcinoma

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PLOS Biology<p><a href="https://fediscience.org/tags/Pancreatic" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>Pancreatic</span></a> ductal <a href="https://fediscience.org/tags/adenocarcinoma" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>adenocarcinoma</span></a> (PDAC) is a deadly <a href="https://fediscience.org/tags/cancer" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>cancer</span></a>. @RoLiefke &amp;co show that chromatin regulator SAMD1 represses EMT-related genes, inhibiting cellular migration, and is downregulated during <a href="https://fediscience.org/tags/PDAC" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>PDAC</span></a> progression by <a href="https://fediscience.org/tags/E3ligase" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>E3ligase</span></a> FBXO11 <a href="https://fediscience.org/tags/PLOSBiology" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>PLOSBiology</span></a> <a href="https://plos.io/4crkx94" rel="nofollow noopener noreferrer" translate="no" target="_blank"><span class="invisible">https://</span><span class="">plos.io/4crkx94</span><span class="invisible"></span></a></p>
PLOS Biology<p>How does <a href="https://fediscience.org/tags/LungCancer" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>LungCancer</span></a> overcome low levels of cholesterol in lung? This study shows that exogenous <a href="https://fediscience.org/tags/cholesterol" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>cholesterol</span></a> can be sensed by lung <a href="https://fediscience.org/tags/adenocarcinoma" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>adenocarcinoma</span></a> cells, activating de novo biosynthesis via GLI2-FOXA3-HMGCS1, promoting cancer progression <a href="https://fediscience.org/tags/PLOSBiology" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>PLOSBiology</span></a> <a href="https://plos.io/3KmW2OM" rel="nofollow noopener noreferrer" translate="no" target="_blank"><span class="invisible">https://</span><span class="">plos.io/3KmW2OM</span><span class="invisible"></span></a></p>
Thomas Vaughan<p>Several studies suggest that bariatric surgery may lower risk of obesity-related cancers, but it&#39;s role in <a href="https://med-mastodon.com/tags/prevention" class="mention hashtag" rel="tag">#<span>prevention</span></a> of <a href="https://med-mastodon.com/tags/esophageal" class="mention hashtag" rel="tag">#<span>esophageal</span></a> <a href="https://med-mastodon.com/tags/cancer" class="mention hashtag" rel="tag">#<span>cancer</span></a> is less clear. A multinational Scandanavian study observed that gastric bypass surgery was associated with a decreased risk of esophageal <a href="https://med-mastodon.com/tags/adenocarcinoma" class="mention hashtag" rel="tag">#<span>adenocarcinoma</span></a> over time after surgery, compared to both the background population and non-operated individuals with morbid obesity. </p><p><a href="https://www.esocan.org/gastric-bypass-surgery-associated-with-reduced-risk-of-esophageal-adenocarcinoma/" target="_blank" rel="nofollow noopener noreferrer" translate="no"><span class="invisible">https://www.</span><span class="ellipsis">esocan.org/gastric-bypass-surg</span><span class="invisible">ery-associated-with-reduced-risk-of-esophageal-adenocarcinoma/</span></a></p><p><a href="https://med-mastodon.com/tags/research" class="mention hashtag" rel="tag">#<span>research</span></a> <a href="https://med-mastodon.com/tags/epidemiology" class="mention hashtag" rel="tag">#<span>epidemiology</span></a></p>
Benjamin S-B :verified:<p>Very exciting that this paper is out now in <a href="https://genomic.social/tags/cancercell" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>cancercell</span></a>: <a href="https://www.cell.com/cancer-cell/fulltext/S1535-6108(23)00216-7" rel="nofollow noopener noreferrer" target="_blank"><span class="invisible">https://www.</span><span class="ellipsis">cell.com/cancer-cell/fulltext/</span><span class="invisible">S1535-6108(23)00216-7</span></a></p><p>In brief: we show that in esophageal <a href="https://genomic.social/tags/adenocarcinoma" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>adenocarcinoma</span></a> <a href="https://genomic.social/tags/checkpoint" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>checkpoint</span></a> <a href="https://genomic.social/tags/therapy" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>therapy</span></a> success is predicted by an independent combination of mutation burden and the amount of <a href="https://genomic.social/tags/monocytes" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>monocytes</span></a> in the tumour pre-treatment (as measured by RNAseq). Very exciting link between <a href="https://genomic.social/tags/myeloid" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>myeloid</span></a> cells and <a href="https://genomic.social/tags/immune" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>immune</span></a> response to <a href="https://genomic.social/tags/cancer" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>cancer</span></a>!</p>
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