David Gregory<p>It will be important to understand how these differences in gene expression contribute to regulation of inflammation and susceptibility to systemic inflammation. Interventions that modulate inflammation to produce a “mouse-like” phenotype could be a powerful new therapeutic strategy for acute inflammatory diseases like <a href="https://med-mastodon.com/tags/sepsis" class="mention hashtag" rel="tag">#<span>sepsis</span></a>, <a href="https://med-mastodon.com/tags/SIRS" class="mention hashtag" rel="tag">#<span>SIRS</span></a>, or <a href="https://med-mastodon.com/tags/shock" class="mention hashtag" rel="tag">#<span>shock</span></a>.</p><p><a href="https://doi.org/10.1186/s10020-024-01052-x" target="_blank" rel="nofollow noopener" translate="no"><span class="invisible">https://</span><span class="ellipsis">doi.org/10.1186/s10020-024-010</span><span class="invisible">52-x</span></a></p><p>/end</p>